| References |
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| REF 1 |
The dynamic alterations of H2AX complex during DNA repair detected by a proteomic approach reveal the critical roles of Ca(2+)/calmodulin in the ionizing radiation-induced cell cycle arrest. Mol Cell Proteomics. 2006 Jun;5(6):1033-44.
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| REF 2 |
Tyrosine phosphorylation of histone H2A by CK2 regulates transcriptional elongation. Nature. 2014 Dec 11;516(7530):267-71.
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| REF 3 |
DNA damage during mitosis invokes a JNK-mediated stress response that leads to cell death. J Cell Biochem. 2010 Jun 1;110(3):725-31.
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| REF 4 |
Functional interaction between FOXO3a and ATM regulates DNA damage response. Nat Cell Biol. 2008 Apr;10(4):460-7.
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| REF 5 |
Vpr-binding protein antagonizes p53-mediated transcription via direct interaction with H3 tail. Mol Cell Biol. 2012 Feb;32(4):783-96.
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| REF 6 |
Mammalian SWI/SNF complexes facilitate DNA double-strand break repair by promoting gamma-H2AX induction. EMBO J. 2006 Sep 6;25(17):3986-97.
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| REF 7 |
FACT-mediated exchange of histone variant H2AX regulated by phosphorylation of H2AX and ADP-ribosylation of Spt16. Mol Cell. 2008 Apr 11;30(1):86-97.
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| REF 8 |
Radiosensitization of esophageal carcinoma cells by the silencing of BMI-1. Oncol Rep. 2016 Jun;35(6):3669-78.
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| REF 9 |
Wild-type p53-induced phosphatase 1 dephosphorylates histone variant gamma-H2AX and suppresses DNA double strand break repair. J Biol Chem. 2010 Apr 23;285(17):12935-47.
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| REF 10 |
Functional interaction between BLM helicase and 53BP1 in a Chk1-mediated pathway during S-phase arrest. J Cell Biol. 2004 Sep 13;166(6):801-13.
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